Tabloski Akunne*
Alcohol consumption has been a pervasive aspect of human culture for millennia, but its detrimental effects on health are increasingly recognized. One such consequence is the accumulation of oxidatively damaged proteins in neural cells and tissues, leading to significant neurobiological repercussions. This article explores the mechanisms by which alcohol-induced oxidative stress contributes to protein damage and aggregation in the nervous system, shedding light on the pathophysiological processes underlying alcohol-related neurodegeneration. Understanding these mechanisms is crucial for developing targeted interventions to mitigate the neurological consequences of alcohol abuse.
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